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New approaches to risk stratification after myocardial infarction.

Journal of cardiovascular pharmacology

Breithardt G, Borggrefe M, Fetsch T, Budde T.
PMID: 1723125
J Cardiovasc Pharmacol. 1991;17:S82-6.

Parameters to assess the presence of electrical instability after myocardial infarction include spontaneous ventricular arrhythmias, late potentials, and programmed ventricular stimulation. The accuracy of the long-term electrocardiogram in correctly identifying high-risk patients has been questioned because spontaneous ventricular arrhythmias...

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Breithardt G, Borggrefe M, Fetsch T, et al. New approaches to risk stratification after myocardial infarction. J Cardiovasc Pharmacol. 1991;17:S82-6
Breithardt, G., Borggrefe, M., Fetsch, T., & Budde, T. (1991). New approaches to risk stratification after myocardial infarction. Journal of cardiovascular pharmacology, 17S82-6.
Breithardt, G, et al. "New approaches to risk stratification after myocardial infarction." Journal of cardiovascular pharmacology vol. 17 (1991): S82-6.
Breithardt G, Borggrefe M, Fetsch T, Budde T. New approaches to risk stratification after myocardial infarction. J Cardiovasc Pharmacol. 1991;17:S82-6. PMID: 1723125.
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Combination Use of Clopidogrel and Proton Pump Inhibitors Increases Major Adverse Cardiovascular Events in Patients With Coronary Artery Disease: A Meta-Analysis.

Journal of cardiovascular pharmacology and therapeutics

Niu Q, Wang Z, Zhang Y, Wang J, Zhang P, Wang C, Yin X, Hou Y.
PMID: 27512080
J Cardiovasc Pharmacol Ther. 2017 Mar;22(2):142-152. doi: 10.1177/1074248416663647. Epub 2016 Aug 20.

BACKGROUND: Published data indicated that combination use of clopidogrel and proton pump inhibitors (PPIs) may increase the incidence of major adverse cardiovascular events (MACEs). This has been a highly controversial topic for years.DESIGN: The present study was performed to...

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Niu Q, Wang Z, Zhang Y, et al. Combination Use of Clopidogrel and Proton Pump Inhibitors Increases Major Adverse Cardiovascular Events in Patients With Coronary Artery Disease: A Meta-Analysis. J Cardiovasc Pharmacol Ther. 2016;22(2):142-152doi: 10.1177/1074248416663647.
Niu, Q., Wang, Z., Zhang, Y., Wang, J., Zhang, P., Wang, C., Yin, X., & Hou, Y. (2017). Combination Use of Clopidogrel and Proton Pump Inhibitors Increases Major Adverse Cardiovascular Events in Patients With Coronary Artery Disease: A Meta-Analysis. Journal of cardiovascular pharmacology and therapeutics, 22(2), 142-152. https://doi.org/10.1177/1074248416663647
Niu, Qiang, et al. "Combination Use of Clopidogrel and Proton Pump Inhibitors Increases Major Adverse Cardiovascular Events in Patients With Coronary Artery Disease: A Meta-Analysis." Journal of cardiovascular pharmacology and therapeutics vol. 22,2 (2017): 142-152. doi: https://doi.org/10.1177/1074248416663647
Niu Q, Wang Z, Zhang Y, Wang J, Zhang P, Wang C, Yin X, Hou Y. Combination Use of Clopidogrel and Proton Pump Inhibitors Increases Major Adverse Cardiovascular Events in Patients With Coronary Artery Disease: A Meta-Analysis. J Cardiovasc Pharmacol Ther. 2017 Mar;22(2):142-152. doi: 10.1177/1074248416663647. Epub 2016 Aug 20. PMID: 27512080.
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Morphological Identification of TRPC7 in Cardiomyocytes from Normal and Renovascular Hypertensive Rats: Retraction.

Journal of cardiovascular pharmacology

[No authors listed]
PMID: 27385186
J Cardiovasc Pharmacol. 2016 Jul;68(1):96. doi: 10.1097/FJC.0000000000000416.

No abstract available.

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Morphological Identification of TRPC7 in Cardiomyocytes from Normal and Renovascular Hypertensive Rats: Retraction. J Cardiovasc Pharmacol. 2016;68(1):96doi: 10.1097/FJC.0000000000000416.
(2016). Morphological Identification of TRPC7 in Cardiomyocytes from Normal and Renovascular Hypertensive Rats: Retraction. Journal of cardiovascular pharmacology, 68(1), 96. https://doi.org/10.1097/FJC.0000000000000416
"Morphological Identification of TRPC7 in Cardiomyocytes from Normal and Renovascular Hypertensive Rats: Retraction." Journal of cardiovascular pharmacology vol. 68,1 (2016): 96. doi: https://doi.org/10.1097/FJC.0000000000000416
Morphological Identification of TRPC7 in Cardiomyocytes from Normal and Renovascular Hypertensive Rats: Retraction. J Cardiovasc Pharmacol. 2016 Jul;68(1):96. doi: 10.1097/FJC.0000000000000416. PMID: 27385186.
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Editorial Page.

Journal of cardiovascular pharmacology and therapeutics

Singh BN.
PMID: 10751308
J Cardiovasc Pharmacol Ther. 2000 Jan;5(1):1. doi: 10.1054/JCPT00500001.

No abstract available.

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Singh BN. Editorial Page. J Cardiovasc Pharmacol Ther. 2000;5(1):1doi: 10.1054/JCPT00500001.
Singh, B. N. (2000). Editorial Page. Journal of cardiovascular pharmacology and therapeutics, 5(1), 1. https://doi.org/10.1054/JCPT00500001
Singh. "Editorial Page." Journal of cardiovascular pharmacology and therapeutics vol. 5,1 (2000): 1. doi: https://doi.org/10.1054/JCPT00500001
Singh BN. Editorial Page. J Cardiovasc Pharmacol Ther. 2000 Jan;5(1):1. doi: 10.1054/JCPT00500001. PMID: 10751308.
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Hydrochlorothiazide Increases Efferent Glomerular Arteriolar Resistance in Spontaneously Hypertensive Rats.

Journal of cardiovascular pharmacology and therapeutics

Komatsu K, Numabe A, Ono Y, Frohlich ED.
PMID: 10684400
J Cardiovasc Pharmacol Ther. 1996 Jan;1(1):57-64. doi: 10.1177/107424849600100109.

BACKGROUND: Micropuncture studies were performed to determine the intrarenal hemodynamic effects of two conventional antihypertensive agents, hydrochlorothiazide (HCTZ) and hydralazine (HYDR) alone and in combination. METHODS AND RESULTS: Male spontaneously hypertensive and normotensive Wistar Kyoto rats (19 weeks old)...

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Komatsu K, Numabe A, Ono Y, et al. Hydrochlorothiazide Increases Efferent Glomerular Arteriolar Resistance in Spontaneously Hypertensive Rats. J Cardiovasc Pharmacol Ther. 1996;1(1):57-64doi: 10.1177/107424849600100109.
Komatsu, K., Numabe, A., Ono, Y., & Frohlich, E. D. (1996). Hydrochlorothiazide Increases Efferent Glomerular Arteriolar Resistance in Spontaneously Hypertensive Rats. Journal of cardiovascular pharmacology and therapeutics, 1(1), 57-64. https://doi.org/10.1177/107424849600100109
Komatsu, et al. "Hydrochlorothiazide Increases Efferent Glomerular Arteriolar Resistance in Spontaneously Hypertensive Rats." Journal of cardiovascular pharmacology and therapeutics vol. 1,1 (1996): 57-64. doi: https://doi.org/10.1177/107424849600100109
Komatsu K, Numabe A, Ono Y, Frohlich ED. Hydrochlorothiazide Increases Efferent Glomerular Arteriolar Resistance in Spontaneously Hypertensive Rats. J Cardiovasc Pharmacol Ther. 1996 Jan;1(1):57-64. doi: 10.1177/107424849600100109. PMID: 10684400.
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Molecular Delivery System for Antisense Oligonucleotides: Enhanced Effectiveness of Antisense Oligonucleotides by HVJ-liposome Mediated Transfer.

Journal of cardiovascular pharmacology and therapeutics

Morishita R, Gibbons GH, Horiuchi M, Nakajima M, Ellison KE, Lee W, Kaneda Y, Ogihara T, Dzau VJ.
PMID: 10684460
J Cardiovasc Pharmacol Ther. 1997 Jul;2(3):213-222. doi: 10.1177/107424849700200308.

BACKGROUND: The effectiveness of antisense oligodeoxynucleotides for in vitro and in vivo studies is limited by a low efficiency of cellular uptake and instability due to degradation by nucleases. To overcome some of these problems, we recently developed a...

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Morishita R, Gibbons GH, Horiuchi M, et al. Molecular Delivery System for Antisense Oligonucleotides: Enhanced Effectiveness of Antisense Oligonucleotides by HVJ-liposome Mediated Transfer. J Cardiovasc Pharmacol Ther. 1997;2(3):213-222doi: 10.1177/107424849700200308.
Morishita, R., Gibbons, G. H., Horiuchi, M., Nakajima, M., Ellison, K. E., Lee, W., Kaneda, Y., Ogihara, T., & Dzau, V. J. (1997). Molecular Delivery System for Antisense Oligonucleotides: Enhanced Effectiveness of Antisense Oligonucleotides by HVJ-liposome Mediated Transfer. Journal of cardiovascular pharmacology and therapeutics, 2(3), 213-222. https://doi.org/10.1177/107424849700200308
Morishita, et al. "Molecular Delivery System for Antisense Oligonucleotides: Enhanced Effectiveness of Antisense Oligonucleotides by HVJ-liposome Mediated Transfer." Journal of cardiovascular pharmacology and therapeutics vol. 2,3 (1997): 213-222. doi: https://doi.org/10.1177/107424849700200308
Morishita R, Gibbons GH, Horiuchi M, Nakajima M, Ellison KE, Lee W, Kaneda Y, Ogihara T, Dzau VJ. Molecular Delivery System for Antisense Oligonucleotides: Enhanced Effectiveness of Antisense Oligonucleotides by HVJ-liposome Mediated Transfer. J Cardiovasc Pharmacol Ther. 1997 Jul;2(3):213-222. doi: 10.1177/107424849700200308. PMID: 10684460.
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The Multicenter Automatic Defibrillator Implantation Trial.

Journal of cardiovascular pharmacology and therapeutics

Epstein AE.
PMID: 10684463
J Cardiovasc Pharmacol Ther. 1997 Jul;2(3):234-237. doi: 10.1177/107424849700200311.

No abstract available.

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Epstein AE. The Multicenter Automatic Defibrillator Implantation Trial. J Cardiovasc Pharmacol Ther. 1997;2(3):234-237doi: 10.1177/107424849700200311.
Epstein, A. E. (1997). The Multicenter Automatic Defibrillator Implantation Trial. Journal of cardiovascular pharmacology and therapeutics, 2(3), 234-237. https://doi.org/10.1177/107424849700200311
Epstein. "The Multicenter Automatic Defibrillator Implantation Trial." Journal of cardiovascular pharmacology and therapeutics vol. 2,3 (1997): 234-237. doi: https://doi.org/10.1177/107424849700200311
Epstein AE. The Multicenter Automatic Defibrillator Implantation Trial. J Cardiovasc Pharmacol Ther. 1997 Jul;2(3):234-237. doi: 10.1177/107424849700200311. PMID: 10684463.
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The Effect of Chloroquine on Circulating Platelet Survival in the Rat.

Journal of cardiovascular pharmacology and therapeutics

Osim E, Mudzingwa S, Musabayane C, Mbajiorgu F, Munjeri O.
PMID: 10684528
J Cardiovasc Pharmacol Ther. 1999 Apr;4(2):97-102. doi: 10.1177/107424849900400204.

BACKGROUND: Chloroquine inhibits platelet aggregation. Because platelet aggregation may lead to the lysis of platelets, the effect of chloroquine administration on circulating platelet survival was studied. METHODS AND RESULTS: Platelets harvested from the blood of male inbred (WAG) rats...

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Osim E, Mudzingwa S, Musabayane C, et al. The Effect of Chloroquine on Circulating Platelet Survival in the Rat. J Cardiovasc Pharmacol Ther. 1999;4(2):97-102doi: 10.1177/107424849900400204.
Osim, E., Mudzingwa, S., Musabayane, C., Mbajiorgu, F., & Munjeri, O. (1999). The Effect of Chloroquine on Circulating Platelet Survival in the Rat. Journal of cardiovascular pharmacology and therapeutics, 4(2), 97-102. https://doi.org/10.1177/107424849900400204
Osim, et al. "The Effect of Chloroquine on Circulating Platelet Survival in the Rat." Journal of cardiovascular pharmacology and therapeutics vol. 4,2 (1999): 97-102. doi: https://doi.org/10.1177/107424849900400204
Osim E, Mudzingwa S, Musabayane C, Mbajiorgu F, Munjeri O. The Effect of Chloroquine on Circulating Platelet Survival in the Rat. J Cardiovasc Pharmacol Ther. 1999 Apr;4(2):97-102. doi: 10.1177/107424849900400204. PMID: 10684528.
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Comparison of the Electromechanical Effects of Vesnarinone and Amrinone in Isolated Dog Purkinje Strands and Ventricular Trabeculae.

Journal of cardiovascular pharmacology and therapeutics

Lathrop DA, Nánási PP, Varró A, Schwartz A.
PMID: 10684410
J Cardiovasc Pharmacol Ther. 1996 Apr;1(2):133-140. doi: 10.1177/107424849600100207.

BACKGROUND: Conventional microelectrode techniques were used to compare the concentration-dependent effects of vesnarinone (0.1-100 µM) and amrinone (1 µM-1 mM) on action potential duration (APD) and developed force in both isolated dog ventricular trabeculae and Purkinje strands. METHODS AND...

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Lathrop DA, Nánási PP, Varró A, et al. Comparison of the Electromechanical Effects of Vesnarinone and Amrinone in Isolated Dog Purkinje Strands and Ventricular Trabeculae. J Cardiovasc Pharmacol Ther. 1996;1(2):133-140doi: 10.1177/107424849600100207.
Lathrop, D. A., Nánási, P. P., Varró, A., & Schwartz, A. (1996). Comparison of the Electromechanical Effects of Vesnarinone and Amrinone in Isolated Dog Purkinje Strands and Ventricular Trabeculae. Journal of cardiovascular pharmacology and therapeutics, 1(2), 133-140. https://doi.org/10.1177/107424849600100207
Lathrop, et al. "Comparison of the Electromechanical Effects of Vesnarinone and Amrinone in Isolated Dog Purkinje Strands and Ventricular Trabeculae." Journal of cardiovascular pharmacology and therapeutics vol. 1,2 (1996): 133-140. doi: https://doi.org/10.1177/107424849600100207
Lathrop DA, Nánási PP, Varró A, Schwartz A. Comparison of the Electromechanical Effects of Vesnarinone and Amrinone in Isolated Dog Purkinje Strands and Ventricular Trabeculae. J Cardiovasc Pharmacol Ther. 1996 Apr;1(2):133-140. doi: 10.1177/107424849600100207. PMID: 10684410.
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Class III Antiarrhythmic Effects of Dofetilide in Rabbit Atrial Myocardium.

Journal of cardiovascular pharmacology and therapeutics

Pallandi RT, Lovell NH, Campbell TJ.
PMID: 10684421
J Cardiovasc Pharmacol Ther. 1996 Jul;1(3):229-234. doi: 10.1053/jcpt.1996.0229.

BACKGROUND: Dofetilide is a new class III antiarrhythmic agent with demonstrated efficacy in ventricular and atrial tachyarrhythmias. We investigated its class III actions and their modulation by stimulation rate in rabbit atrial myocardium. METHODS AND RESULTS: Standard microelectrode techniques...

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Pallandi RT, Lovell NH, Campbell TJ. Class III Antiarrhythmic Effects of Dofetilide in Rabbit Atrial Myocardium. J Cardiovasc Pharmacol Ther. 1996;1(3):229-234doi: 10.1053/jcpt.1996.0229.
Pallandi, R. T., Lovell, N. H., & Campbell, T. J. (1996). Class III Antiarrhythmic Effects of Dofetilide in Rabbit Atrial Myocardium. Journal of cardiovascular pharmacology and therapeutics, 1(3), 229-234. https://doi.org/10.1053/jcpt.1996.0229
Pallandi, et al. "Class III Antiarrhythmic Effects of Dofetilide in Rabbit Atrial Myocardium." Journal of cardiovascular pharmacology and therapeutics vol. 1,3 (1996): 229-234. doi: https://doi.org/10.1053/jcpt.1996.0229
Pallandi RT, Lovell NH, Campbell TJ. Class III Antiarrhythmic Effects of Dofetilide in Rabbit Atrial Myocardium. J Cardiovasc Pharmacol Ther. 1996 Jul;1(3):229-234. doi: 10.1053/jcpt.1996.0229. PMID: 10684421.
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Electrophysiology of Myocardial Cells in the Epicardial, Midmyocardial, and Endocardial Layers of the Ventricle.

Journal of cardiovascular pharmacology and therapeutics

Lukas A.
PMID: 10684443
J Cardiovasc Pharmacol Ther. 1997 Jan;2(1):61-72. doi: 10.1177/107424849700200108.

The recent discovery of multiple myocardial cell types in the ventricular wall of most species has prompted a reevaluation of several electrophysiologic and electrocardiographic findings. This review briefly presents the salient electrophysiologic features of myocardial cells in the epicardial,...

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Lukas A. Electrophysiology of Myocardial Cells in the Epicardial, Midmyocardial, and Endocardial Layers of the Ventricle. J Cardiovasc Pharmacol Ther. 1997;2(1):61-72doi: 10.1177/107424849700200108.
Lukas, A. (1997). Electrophysiology of Myocardial Cells in the Epicardial, Midmyocardial, and Endocardial Layers of the Ventricle. Journal of cardiovascular pharmacology and therapeutics, 2(1), 61-72. https://doi.org/10.1177/107424849700200108
Lukas. "Electrophysiology of Myocardial Cells in the Epicardial, Midmyocardial, and Endocardial Layers of the Ventricle." Journal of cardiovascular pharmacology and therapeutics vol. 2,1 (1997): 61-72. doi: https://doi.org/10.1177/107424849700200108
Lukas A. Electrophysiology of Myocardial Cells in the Epicardial, Midmyocardial, and Endocardial Layers of the Ventricle. J Cardiovasc Pharmacol Ther. 1997 Jan;2(1):61-72. doi: 10.1177/107424849700200108. PMID: 10684443.
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Nitric Oxide Synthesis Inhibition and Role of P-selectin in Leukocyte Adhesion to Vascular Tissues.

Journal of cardiovascular pharmacology and therapeutics

Yang BC, Mehta P, Mehta JL.
PMID: 10684448
J Cardiovasc Pharmacol Ther. 1997 Apr;2(2):107-114. doi: 10.1177/107424849700200204.

BACKGROUND: This study was designed to examine the role of P-selectin expression in leukocyte adhesion to endothelium caused by inhibition of nitric oxide synthesis. METHODS AND RESULTS: Rat aortic rings were treated with the nitric oxide synthesis inhibitor N(omicron)-nitro-l-arginine...

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Yang BC, Mehta P, Mehta JL. Nitric Oxide Synthesis Inhibition and Role of P-selectin in Leukocyte Adhesion to Vascular Tissues. J Cardiovasc Pharmacol Ther. 1997;2(2):107-114doi: 10.1177/107424849700200204.
Yang, B. C., Mehta, P., & Mehta, J. L. (1997). Nitric Oxide Synthesis Inhibition and Role of P-selectin in Leukocyte Adhesion to Vascular Tissues. Journal of cardiovascular pharmacology and therapeutics, 2(2), 107-114. https://doi.org/10.1177/107424849700200204
Yang, et al. "Nitric Oxide Synthesis Inhibition and Role of P-selectin in Leukocyte Adhesion to Vascular Tissues." Journal of cardiovascular pharmacology and therapeutics vol. 2,2 (1997): 107-114. doi: https://doi.org/10.1177/107424849700200204
Yang BC, Mehta P, Mehta JL. Nitric Oxide Synthesis Inhibition and Role of P-selectin in Leukocyte Adhesion to Vascular Tissues. J Cardiovasc Pharmacol Ther. 1997 Apr;2(2):107-114. doi: 10.1177/107424849700200204. PMID: 10684448.
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Showing 1 to 12 of 633 entries